Etiology of cerebral palsy
Gespeichert in:
Verfasser / Beitragende:
[Alf Meberg, Harald Broch]
Ort, Verlag, Jahr:
2004
Enthalten in:
Journal of Perinatal Medicine, 32/5(2004-09-01), 434-439
Format:
Artikel (online)
Online Zugang:
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| 024 | 7 | 0 | |a 10.1515/JPM.2004.143 |2 doi |
| 035 | |a (NATIONALLICENCE)gruyter-10.1515/JPM.2004.143 | ||
| 245 | 0 | 0 | |a Etiology of cerebral palsy |h [Elektronische Daten] |c [Alf Meberg, Harald Broch] |
| 520 | 3 | |a Aims: To register the prevalence of cerebral palsy (CP) and determine etiological factors for the condition. Methods: Population based study with registration of CP-cases in children born during the 30-year period 1970-99. Cases with postneonatal etiology were excluded. Results: 166 CP-cases were registered among 70 824 children, a prevalence of 2.3 per 1000 live born infants. The prevalence did not change significantly during the period. 66 (40%) were low birthweight infants (LBWIs; <2500 g), and 100 (60%) normal birthweight infants (NBWIs; ≥2500 g). The origin was classified as prenatal in 37 (22%), perinatal/neonatal in 78 (47%) and unclassifiable in 51 (31%). In LBWIs 39/66 (59%) had a perinatal/neonatal etiology, most frequently intra- or periventricular hemorrhages (IVH/PVH) and/or periventricular leukomalacia (PVL) or cerebral infarctions (CI) (17; 44%). In NBWIs 39/100 (39%) had a perinatal etiology, most frequently hypoxic-ischemic encephalopathy (HIE) (31; 79%). Conclusions: In a substantial percentage of CP-cases perinatal/neonatal brain injury was classified as the cause. Among these IVH/PVH/PVL/CI dominated in LBWIs, while HIE dominated in NBWIs. Our data may point to preventability of a larger part of CP than earlier suggested. | |
| 540 | |a © Walter de Gruyter | ||
| 690 | 7 | |a Human reproduction, growth & development |2 nationallicence | |
| 690 | 7 | |a Gynaecology & obstetrics |2 nationallicence | |
| 690 | 7 | |a Paediatric medicine |2 nationallicence | |
| 690 | 7 | |a Cerebral infarction |2 nationallicence | |
| 690 | 7 | |a cerebral palsy |2 nationallicence | |
| 690 | 7 | |a etiological factors |2 nationallicence | |
| 690 | 7 | |a neonatal encephalopathy |2 nationallicence | |
| 690 | 7 | |a neonatal mortality |2 nationallicence | |
| 690 | 7 | |a periventricular hemorrhage |2 nationallicence | |
| 690 | 7 | |a periventricular leukomalacia |2 nationallicence | |
| 700 | 1 | |a Meberg |D Alf |u Department of Paediatrics, Vestfold Hospital Tønsberg, Norway |4 aut | |
| 700 | 1 | |a Broch |D Harald |u Department of Paediatrics, Vestfold Hospital Tønsberg, Norway |4 aut | |
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| 950 | |B NATIONALLICENCE |P 700 |E 1- |a Meberg |D Alf |u Department of Paediatrics, Vestfold Hospital Tønsberg, Norway |4 aut | ||
| 950 | |B NATIONALLICENCE |P 700 |E 1- |a Broch |D Harald |u Department of Paediatrics, Vestfold Hospital Tønsberg, Norway |4 aut | ||
| 950 | |B NATIONALLICENCE |P 773 |E 0- |t Journal of Perinatal Medicine |d Walter de Gruyter |g 32/5(2004-09-01), 434-439 |x 0300-5577 |q 32:5<434 |1 2004 |2 32 |o jpme | ||
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