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   <subfield code="a">10.1007/s00421-006-0156-2</subfield>
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   <subfield code="a">Influence of lung volume, glossopharyngeal inhalation and P ETO2 and P ETCO2 on apnea performance in trained breath-hold divers</subfield>
   <subfield code="h">[Elektronische Daten]</subfield>
   <subfield code="c">[Kristian Overgaard, Søren Friis, Rasmus Pedersen, Gunnar Lykkeboe]</subfield>
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   <subfield code="a">Breath-hold divers train and compete in maximal apnea performance. Glossopharyngeal inhalation (GI) is commonly used to increase lung volume above vital capacity (VC) prior to apnea. We investigated the hypothesis that this practice would increase apnea performance and relaxed airway pressure. Seven well-trained breath-hold divers performed maximal bouts of apnea at three different lung volumes (85% VC, VC and VC + GI) both at rest (dry static apnea) and during underwater swimming (dynamic apnea). Heart rate, apnea time and end tidal PCO2 and PO2 (P ETCO2 and P ETO2) were recorded. In addition, relaxed airway pressure was measured after GI. Maximal GI increased lung volume by 1.59±0.57l above VC and increased relaxed airway pressure to from 3.5±0.5 to 8.7±1.7kPa. Dry static apnea time was higher at VC + GI (346±46s) than at VC (309±38s, P&lt;0.05) and 85% VC (297±48s, P&lt;0.01). Likewise, dynamic apnea time was higher at VC + GI (97±27s) than at VC (78±14s, P&lt;0.05) and 85% VC (71±17s, P&lt;0.05). P ETO2 values reached 3.5±0.6kPa at the end of dry static apnea bouts and this was not different from dynamic apnea when taking hydrostatic pressure at swimming depth into account (3.7±0.6kPa, P=0.48). In conclusion, GI increases lung volume, relaxed airway pressure and apnea performance in well-trained breath-hold divers.</subfield>
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   <subfield code="a">Springer-Verlag, 2006</subfield>
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  <datafield tag="690" ind1=" " ind2="7">
   <subfield code="a">Lung packing</subfield>
   <subfield code="2">nationallicence</subfield>
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   <subfield code="a">Lung compliance</subfield>
   <subfield code="2">nationallicence</subfield>
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   <subfield code="a">Hypoxia</subfield>
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   <subfield code="a">Overgaard</subfield>
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   <subfield code="u">Department of Sport Science, University of Aarhus, Katrinebjergvej 89C, 8200, Aarhus N, Denmark</subfield>
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   <subfield code="a">Friis</subfield>
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   <subfield code="a">Pedersen</subfield>
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   <subfield code="u">Department of Sport Science, University of Aarhus, Katrinebjergvej 89C, 8200, Aarhus N, Denmark</subfield>
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   <subfield code="u">Department of Zoophysiology, University of Aarhus, C.F. Møllers Allé 131, Universitetsparken, 8000, Aarhus C, Denmark</subfield>
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   <subfield code="t">European Journal of Applied Physiology</subfield>
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   <subfield code="g">97/2(2006-05-01), 158-164</subfield>
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