caa a22 4500 469090677 CHVBK 20180323133016.0 cr unu---uuuuu 170328e19920601xx s 000 0 eng 10.1007/BF00464701 doi (NATIONALLICENCE)springer-10.1007/BF00464701 Horsfall A. Kennedy Institute of Rheumatology, Bute Gardens, W6 7DW, London, UK aut Molecular mimicry and autoantigens in connective tissue diseases [Elektronische Daten] [A. Horsfall] Conclusion: Unless the basis of reactivity can be strictly elucidated the biological significance of molecular mimicry must be held in question and the attention given to fulfilling the criteria outlined in table 2 cannot be overstressed. Nevertheless, the evidence so far is intriguing in implicating some form of role for retroviruses or bacteria in certain autoimmune diseases. The reader would be well advised to keep an open mind and await with critical interest more stringent data from those laboratories with the resources, patience and dedication to pursue this line of research. Kluwer Academic Publishers, 1992 autoantigen nationallicence connective tissue disease nationallicence molecular mimicry nationallicence Molecular Biology Reports Kluwer Academic Publishers 16/3(1992-06-01), 139-147 0301-4851 16:3<139 1992 16 11033 https://doi.org/10.1007/BF00464701 text/html Onlinezugriff via DOI 1 research-article jats NATIONALLICENCE 856 40 https://doi.org/10.1007/BF00464701 text/html Onlinezugriff via DOI NATIONALLICENCE 100 1- Horsfall A. Kennedy Institute of Rheumatology, Bute Gardens, W6 7DW, London, UK aut NATIONALLICENCE 773 0- Molecular Biology Reports Kluwer Academic Publishers 16/3(1992-06-01), 139-147 0301-4851 16:3<139 1992 16 11033 Metadata rights reserved Springer special CC-BY-NC licence nationallicence BK010053 XK010053 XK010000 NATIONALLICENCE NATIONALLICENCE NL-springer