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   <subfield code="a">Asynchronization of local population dynamics and persistence of a metapopulation: a lesson from an endangered composite plant, Aster kantoensis</subfield>
   <subfield code="h">[Elektronische Daten]</subfield>
   <subfield code="c">[M. Shimada, F. Ishihama]</subfield>
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   <subfield code="a">Fragmentation of a large habitat makes local populations less linked to others, and a whole population structure changes to a metapopulation. The smaller a local population is, the more strengthened extinction factors become. Then, frequent extinctions of local populations threaten persistence of the metapopulation unless recolonizations occur rapidly enough after local extinctions. Spatially structured models have been more widely used for predicting future population dynamics and for assessing the extinction risk of a metapopulation. In this article, we first review such spatially structured models that have been applied to conservation biology, focusing on effects of asynchronization among local population dynamics on persistence of the whole metapopulation. Second, we introduce our ongoing project on extinction risk assessment of an endangered composite biennial plant, Aster kantoensis, in the riverside habitat, based on a lattice model for describing its spatiotemporal population dynamics. The model predicted that the extinction risk of A. kantoensis depends on both the frequency of flood occurrence and the time to coverage of a local habitat by other competitively stronger perennials. Finally, we present a measure (Hassell and Pacala's CV 2) for quantifying the effect of asynchronization among local population dynamics on the persistence of a whole metapopulation in conservation ecology.</subfield>
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   <subfield code="a">The Society of Population Ecology and Springer-Verlag Tokyo, 2000</subfield>
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   <subfield code="a">Key words Habitat fragmentation</subfield>
   <subfield code="2">nationallicence</subfield>
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   <subfield code="a">Spatially structured model</subfield>
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   <subfield code="a">Extinction risk assessment</subfield>
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   <subfield code="a">Shimada</subfield>
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   <subfield code="u">Department of Systems Sciences (Biology), University of Tokyo, 3-8-1 Komaba, Meguro, Tokyo 153-8902, Japan Tel. +81-3-5454-6796; Fax +81-3-5454-6998 e-mail: mshimada@balmer.c.u-tokyo.ac.jp, JP</subfield>
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   <subfield code="a">Ishihama</subfield>
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   <subfield code="u">Department of Systems Sciences (Biology), University of Tokyo, 3-8-1 Komaba, Meguro, Tokyo 153-8902, Japan Tel. +81-3-5454-6796; Fax +81-3-5454-6998 e-mail: mshimada@balmer.c.u-tokyo.ac.jp, JP</subfield>
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   <subfield code="u">Department of Systems Sciences (Biology), University of Tokyo, 3-8-1 Komaba, Meguro, Tokyo 153-8902, Japan Tel. +81-3-5454-6796; Fax +81-3-5454-6998 e-mail: mshimada@balmer.c.u-tokyo.ac.jp, JP</subfield>
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   <subfield code="u">Department of Systems Sciences (Biology), University of Tokyo, 3-8-1 Komaba, Meguro, Tokyo 153-8902, Japan Tel. +81-3-5454-6796; Fax +81-3-5454-6998 e-mail: mshimada@balmer.c.u-tokyo.ac.jp, JP</subfield>
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   <subfield code="a">Metadata rights reserved</subfield>
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