caa a22 4500 605458138 CHVBK 20210128100227.0 cr unu---uuuuu 210128e20150301xx s 000 0 eng 10.1007/s00167-013-2512-1 doi (NATIONALLICENCE)springer-10.1007/s00167-013-2512-1 Compensatory muscle activation caused by tendon lengthening post-Achilles tendon rupture [Elektronische Daten] [Stephen Suydam, Thomas Buchanan, Kurt Manal, Karin Silbernagel] Purpose: The purpose of this study was to establish a relationship between the lengthening of the Achilles tendon post-rupture and surgical repair to muscle activation patterns during walking in order to serve as a reference for post-surgical assessment. Method: The Achilles tendon lengths were collected from 4 patients with an Achilles tendon rupture 6 and 12months post-surgery along with 5 healthy controls via ultrasound. EMG was collected from the triceps surae muscles and tibialis anterior during overground walking. Results: Achilles lengths at 6 and 12months post-surgery were significantly longer (p<0.05) on the involved side compared to the uninvolved side, but there were no side-to-side differences in the healthy controls. The integrated EMG (iEMG) of the involved side was significantly higher than the uninvolved side in the lateral gastrocnemius at 6months and for the medial gastrocnemius at 12months in the patients with Achilles tendon rupture; no side-to-side difference was found in the healthy controls. The triceps surae muscles' activations were fair to moderately correlated to the Achilles lengths (0.38<r<0.52). Conclusions: The increased Achilles tendon length and iEMG from the triceps surae muscles indicate that loss of function is primarily caused by anatomical changes in the tendon and the appearance of muscle weakness is due to a lack of force transmission capability. This study indicates that when aiming for full return of function and strength, an important treatment goal appears to be to minimize tendon elongation. Level of evidence: Prognostic prospective case series, Level IV. Springer-Verlag Berlin Heidelberg, 2013 EMG nationallicence Gait nationallicence Neural inhibition nationallicence Surgery nationallicence Ankle nationallicence Suydam Stephen University of Delaware, 126 Spencer Lab, 19716-3140, Newark, DE, USA aut Buchanan Thomas University of Delaware, 126 Spencer Lab, 19716-3140, Newark, DE, USA aut Manal Kurt University of Delaware, 126 Spencer Lab, 19716-3140, Newark, DE, USA aut Silbernagel Karin University of the Sciences, 600 S. 43rd Street, 19104, Philadelphia, PA, USA aut Knee Surgery, Sports Traumatology, Arthroscopy Springer Berlin Heidelberg 23/3(2015-03-01), 868-874 0942-2056 23:3<868 2015 23 167 https://doi.org/10.1007/s00167-013-2512-1 text/html Onlinezugriff via DOI BK010053 XK010053 XK010000 Metadata rights reserved Springer special CC-BY-NC licence nationallicence 1 research-article jats NATIONALLICENCE NATIONALLICENCE NL-springer NATIONALLICENCE 856 40 https://doi.org/10.1007/s00167-013-2512-1 text/html Onlinezugriff via DOI NATIONALLICENCE 700 1- Suydam Stephen University of Delaware, 126 Spencer Lab, 19716-3140, Newark, DE, USA aut NATIONALLICENCE 700 1- Buchanan Thomas University of Delaware, 126 Spencer Lab, 19716-3140, Newark, DE, USA aut NATIONALLICENCE 700 1- Manal Kurt University of Delaware, 126 Spencer Lab, 19716-3140, Newark, DE, USA aut NATIONALLICENCE 700 1- Silbernagel Karin University of the Sciences, 600 S. 43rd Street, 19104, Philadelphia, PA, USA aut NATIONALLICENCE 773 0- Knee Surgery, Sports Traumatology, Arthroscopy Springer Berlin Heidelberg 23/3(2015-03-01), 868-874 0942-2056 23:3<868 2015 23 167